LncRNA Chaer prevents cardiomyocyte apoptosis from acute myocardial infarction through AMPK activation

Z He, X Zeng, D Zhou, P Liu, D Han, L Xu… - Frontiers in …, 2021 - frontiersin.org
Long non-coding RNA (lncRNA) is widely reported to be involved in cardiac (patho)
physiology. Acute myocardial infarction, in which cardiomyocyte apoptosis plays an …

[PDF][PDF] Serum levels of high-sensitivity troponin T: a novel marker for left ventricular remodeling and performance in hypertensive subjects

DM Miao, LP Zhang, HP Yu, JY Zhang, WK Xiao… - Genet Mol …, 2014 - funpecrp.com.br
It has been well established that high-sensitivity cardiac troponin T (hs-TnT) is a specific and
highly sensitive marker in acute coronary syndromes. On the other hand, studies on serum …

Modulation of staurosporine-activated volume-sensitive outwardly rectifying Cl-channel by PI3K/Akt in cardiomyocytes

Y Liu, B Wang, WW Zhang, JN Liu… - Current …, 2013 - ingentaconnect.com
Chloride (Cl-) channels participate in the regulation of cardiac function in response to stress
although the underlying regulatory mechanism remains poorly understood. This study was …

The coexistence of nocturnal sustained hypoxia and obesity additively increases cardiac apoptosis

SD Lee, WW Kuo, DT Bau, FY Ko… - Journal of Applied …, 2008 - journals.physiology.org
Background: nocturnal sustained hypoxia during sleeping time has been reported in severe
obesity, but no information regarding the cardiac molecular mechanism in the coexistence of …

Sacubitril/valsartan reduces levels of procollagen types I and III and correlates with reverse cardiac remodeling

CA Plata-Mosquera, W Bernal-Tórres… - REC …, 2021 - Elsevier
Introduction and objectives Extracellular matrix turnover is a major determinant of cardiac
remodeling in heart failure. Sacubitril/valsartan decreases serum levels of procollagen type I …

Role of calcineurin in Porphyromonas gingivalis-induced myocardial cell hypertrophy and apoptosis

SD Lee, WW Kuo, DY Lin, TH Chen, WH Kuo… - Journal of biomedical …, 2006 - Springer
Background and objective Periodontal pathogen Porphyromonas gingivalis (P. gingivalis)
increased cardiomyocyte hypertrophy and apoptosis whereas Actinobaeillus …

P38 mitogen‐activated protein kinase pathways are involved in the hypertrophy and apoptosis of cardiomyocytes induced by Porphyromonas gingivalis conditioned …

HC Wu, YL Yeh, WW Kuo, SK Huang… - … biochemistry and its …, 2008 - Wiley Online Library
The surrounding medium of periodontal pathogen Porphyromonas gingivalis (P. gingivalis)
increased cardiomyocyte hypertrophy and apoptosis whereas Actinobaeillus …

Changes in Important Apoptosis-Related Molecules in the Endothelin-1–Induced Hypertrophied Cardiomyocytes: Effect of the Pretreatment with Eicosapentaenoic …

N Shimojo, S Jesmin, S Zaedi… - Experimental …, 2006 - journals.sagepub.com
Human heart failure is preceded by a process called cardiac remodeling, in which heart
chambers progressively enlarge and contractile function deteriorates. Programmed cell …

Role of mitogen‐activated protein kinase kinase in Porphyromonas gingivalis‐induced myocardial cell hypertrophy and apoptosis

SD Lee, SH Chang, WH Kuo, TH Ying… - European journal of …, 2006 - Wiley Online Library
Secreted factors present in the medium following growth of the periodontal pathogen
Porphyromonas gingivalis cause increased cardiomyocyte hypertrophy and apoptosis …

Novel imaging strategies for predicting remodeling and evolution of heart failure: targeting the renin-angiotensin system

J Shirani, J Narula, WC Eckelman… - Heart Failure …, 2006 - heartfailure.theclinics.com
Left ventricular (LV) remodeling constitutes the final common pathway for the progression of
systolic dysfunction to end-stage heart failure (HF)[1, 2]. The renin–angiotensin system …