This narrative review summarises the evidence for considering physical exercise (PE) as a
non-pharmacological intervention for delaying cognitive decline in patients with Alzheimer's …

The field of vascular contributions to cognitive impairment and dementia (VCID) is evolving
rapidly. Research in VCID encompasses topics aiming to understand, prevent, and treat the …

Background Alzheimer's disease (AD) is notably associated with cognitive decline resulting
from impaired function of hippocampal and cortical areas; however, several other domains …

Regular exercise has been shown to reduce the risk of Alzheimer's disease (AD). Our
previous study showed that the protein aquaporin 4 (AQP4), which is specifically expressed …

Background Hypothalamic dysfunction occurs early in the clinical course of Alzheimer's
disease (AD), likely contributing to disturbances in feeding behavior and metabolic function …

Abstract CYLD lysine 63 deubiquitinase (CYLD), that is mainly involved in immune
responses and inflammation, is expressed at high levels in the brain, especially in the dorsal …

Physical exercise has been suggested to reduce the risk of developing Alzheimer's disease
(AD) as well as ameliorate the progression of the disease. However, we recently published …

Single-factor intervention, such as physical exercise and auditory and visual stimulation,
plays a positive role on the prevention and treatment of Alzheimer's disease (AD); however …

Cerebral amyloid angiopathy (CAA) is a small vessel disease characterized by amyloid β
(Aβ) peptide deposition within the walls of medium to small-caliber blood vessels, cerebral …

Cerebral amyloid angiopathy (CAA) is the deposition of amyloid protein in the cerebral
vasculature, a common feature in both aging and Alzheimer's disease (AD). However, the …