Oxytocin contributes to the regulation of cytoskeletal and synaptic proteins and could,
therefore, affect the mechanisms of neurodevelopmental disorders, including autism. Both …

Abstract In Prader-Willi syndrome (PWS), obesity is caused by the disruption of appetite-
controlling pathways in the brain. Two PWS candidate genes encode MAGEL2 and necdin …

Individuals with Prader-Willi syndrome (PWS) display developmental delays, cognitive
impairment, excessive hunger, obesity, and various behavioral abnormalities. Current PWS …

Genomic imprinting is a term used for an intergenerational epigenetic inheritance and
involves a subset of genes expressed in a parent-of-origin-dependent way. Imprinted genes …

Background Responding to social signals by expressing the correct behavior is not only
challenged in autism but also in diseases with a high prevalence of autism, such as Prader …

Maternal separation (MS) is known to induce permanent changes in the central nervous
system and is associated with increased levels of anxiety and cognitive impairments. The …

MAGEL2 is one of five protein‐coding, maternally imprinted, paternally expressed genes in
the Prader–Willi syndrome (PWS)‐critical domain on chromosome 15q11‐q13. Truncating …

The hypothalamic neuropeptide oxytocin (OT) is a forefront molecule among neuropeptides
due to its pronounced prosocial effects and its potential use in socioemotional deficits that …

Oxytocin and vasopressin systems have been studied separately in autism spectrum
disorder (ASD). Here, we provide evidence from an evolutionary and neuroscience …

Prader–Willi syndrome (PWS) is a genetic disorder characterized by a variety of
physiological and behavioral dysregulations, including hyperphagia, a condition that can …