Background The spinal cord is an important anatomic site at which volatile agents act to
prevent movement in response to a noxious stimulus. This study was designed to test the …

Developmental ethanol exposure damages the hippocampus, causing long-lasting learning
and memory deficits. Synaptic plasticity mechanisms (eg, long-term potentiation [LTP]) …

Ethanol is a general anesthetic agent as defined by abolition of movement in response to
noxious stimulation. This anesthetic endpoint is due to spinal anesthetic actions. This study …

Initial sensitivity and acute tolerance to ethanol have been implicated as risk factors in the
development of alcoholism in humans. These behaviors were investigated in rats selectively …

Ethanol (EtOH) damages the hippocampus, a brain region that is involved in learning and
memory processes. The mechanisms responsible for this effect of EtOH are not fully …

Ethanol (EtOH) tachyphylaxis (acute tolerance), a time‐dependent decrease in apparent
potency, is known in vivo and in some neuronal preparations. The present studies …

Background Gamma-aminobutyric acid type A (GABA (A)) receptors are considered
important in mediating anesthetic actions. Mice lacking the beta3 subunit of this receptor …

El presente trabajo se propuso analizar la utilidad de una planilla que registra los cambios
clínicos para caracterizar la sedoanalgesia obtenida con ketamina-midazolam-fentanilo en …

El objetivo del estudio consistió en modelizar, a través de agrupamientos multivariados, la
respuesta a un método de sedoanalgesia con halotano efectuado en ratones, mediante …