In animal models, prolonged exposure (2 h) to high-level noise causes an irreparable
damage to the synapses between the inner hair cells and auditory nerve fibers within the …

Purpose: Animal models and human temporal bones indicate that noise exposure is a risk
factor for cochlear synaptopathy, a possible etiology of tinnitus. Veterans are exposed to …

Higher stages of central auditory processing compensate for a loss of cochlear nerve
synapses by increasing the gain on remaining afferent inputs, thereby restoring firing rate …

Many tinnitus patients report difficulties understanding speech in noise or competing talkers,
despite having “normal” hearing in terms of audiometric thresholds. The interference caused …

Age-related hearing loss is the most prevalent sensory impairment in the older adult
population and is related to noise-induced damage or age-related deterioration of the …

Sensory hair cells in the cochlea transduce environmental sounds into driven high-fidelity
neural signals that are conveyed by auditory nerve fibers to structures in the central auditory …

In this position review, we propose to establish a path for replacing the empirical
classification of tinnitus with a taxonomy from precision medicine. The goal of a classification …

Hearing research findings in recent years have begun to change how we think about
hearing loss and how we consider the risk of auditory damage from noise exposure. These …

Introduction Accumulating evidence suggests a role of the brainstem in tinnitus generation
and modulation. Several studies in chronic tinnitus patients have reported latency and …

Purpose Growing evidence supports the inclusion of perceptual tests that quantify the
processing of temporal fine structure (TFS) in clinical hearing assessment. Many tasks have …