The roles of both neuroinflammation and oxidative stress in the pathophysiology of epilepsy
have begun to receive considerable attention in recent years. However, these concepts are …

CD56+ T cells are generally recognized as a distinct population of T cells and are
categorized as NKT-like cells. Although our understanding of NKT-like cells is far from …

Febrile Infection-Related Epilepsy Syndrome (FIRES) is a unique catastrophic epilepsy
syndrome, and the development of drug-resistant epilepsy (DRE) is inevitable. Recently …

Pericytes are a component of the blood–brain barrier (BBB) neurovascular unit, in which
they play a crucial role in BBB integrity and are also implicated in neuroinflammation. The …

Accumulating evidence has demonstrated that the pathogenesis of epilepsy is linked to
neuroinflammation and cerebrovascular dysfunction. Peripheral immune cell invasion into …

Objective Therapeutic strategies for patients with febrile infection‐related epilepsy syndrome
(FIRES) are limited, ad hoc, and frequently ineffective. Based on evidence that inflammation …

We analyzed peripheral blood mononuclear cells (PBMCs) and serum inflammatory
biomarkers in patients with mesial temporal lobe epilepsy (drug-resistant–DR, vs. drug …

Neuroinflammation has been implicated in the pathogenesis of West syndrome (WS).
Inflammatory cytokines, including interleukin-1β (IL-1β), have been reported to be …

Background: Inflammation, particularly cytokine release, contributes to epileptogenesis by
influencing the cerebral tissue remodeling and neuronal excitability that occurs after a …

Abstract Background West Syndrome (WS) is an epileptic encephalopathy that typically
occurs in infants and is characterized by hypsarrhythmia, infantile spasms, and …