Postprandial release of the incretin glucagon-like peptide-1 (GLP-1) has been suggested to
act as an endogenous satiety factor in humans. In rats, however, the evidence for this is …

The hormone glucagon‐like peptide‐1 (GLP‐1) is released from the gut in response to food
intake. It acts as a satiety signal, leading to reduced food intake, and also as a regulator of …

Exogenous activation of central nervous system glucagon-like peptide-1 (GLP-1) receptors
(GLP-1Rs) reduces food intake. Experiments addressed whether endogenous central GLP …

Glucagon-like peptide-1 (GLP-1) is released in response to meals and exerts important roles
in the maintenance of normal glucose homeostasis. GLP-1 is also important in the regulation …

The signaling systems underlying eating behavior control are complex. The current review
focuses on gastrointestinal (GI) signaling systems as physiological key functions for …

This study tested the hypothesis that systemic stressors in rats activate glucagon-like peptide-
1 (GLP-1)-immunoreactive neurons in the caudal brain stem, including those that project to …

Recent research has indicated that appetite-regulating hormones from the gut may have
therapeutic potential. The incretin hormone, glucagon-like peptide-1 (GLP-1), appears to be …

Therapies targeting the action of incretin hormones have been under close scrutiny in recent
years. The incretin effect has been defined as postprandial enhancement of insulin secretion …

Glucagon-like peptide-1 (GLP-1) relaxes the stomach during fasting but decreases hunger
and food consumption and retards gastric emptying. The interrelationships between volume …

Recent studies have indicated that GIP and GLP-1 are about as important as each other in
the incretin effect, being released rapidly after meals and being active already at fasting …