Myocardial ischemia-reperfusion injury (MIRI) results in the aggravation of myocardial injury
caused by rapid recanalization of the ischemic myocardium. In the past few years, there is a …

Extracellular vesicles (EVs), through their complex cargo, can reflect the state of their cell of
origin and change the functions and phenotypes of other cells. These features indicate …

Extracellular vesicles (EVs) can regulate the polarization of macrophages in a variety of
inflammatory diseases by mediating intercellular signal transduction and affecting the …

Cancer‐associated fibroblasts (CAFs) as a predominant cell component in the tumour
microenvironment (TME) play an essential role in tumour progression. Our earlier studies …

Ferroptosis is a predominant contributor to renal ischemia reperfusion injury (IRI) after
kidney transplant, evoking delayed graft function and poorer long-term outcomes. The wide …

The overproduction of reactive oxygen species (ROS) and burst of inflammation following
cardiac ischemia–reperfusion (I/R) are the leading causes of cardiomyocyte injury …

Obesity, particularly increased visceral fat, positively correlates with various metabolic
challenges, including atherosclerosis, but the mechanism is not fully understood. The aim of …

Cardiomyocyte apoptosis in response to inflammation is a primary cause of myocardial
ischemia-reperfusion injury (IRI). Nuclear factor erythroid 2 like 2 (Nrf2) reportedly plays an …

Myocardial ischemia/reperfusion (I/R) injury is the resultantly pathological heart damage
from the restoration of blood supply through percutaneous coronary intervention. Herein …

Myocardial infarction is a major cause of morbidity and mortality worldwide. Due to poor
inherent regeneration of the adult mammalian myocardium and challenges with effective …