Tim-3 cell signaling and iNOS are involved in the protective effects of ischemic postconditioning against focal ischemia in rats
The protective effect of ischemic postconditioning (IPostC) against stroke has been well-
established, and the underlying mechanisms are known to involve inhibited-inflammation …
established, and the underlying mechanisms are known to involve inhibited-inflammation …
Effect of ischemic postconditioning on cerebral edema and the AQP4 expression following hypoxic-eschemic brain damage in neonatal rats
L Yu, SJ Fan, L Liu, M Xiao, XJ Lin, Y Liu, HX Lv… - World Journal of …, 2015 - Springer
Background A rat model for neonatal hypoxic-ischemic brain damage (HIBD) was
established to observe the effect of ischemic postconditioning (IPostC) on cerebral edema …
established to observe the effect of ischemic postconditioning (IPostC) on cerebral edema …
Timing of xenon-induced delayed postconditioning to protect against spinal cord ischaemia–reperfusion injury in rats
YW Yang, WP Cheng, JK Lu, XH Dong… - British Journal of …, 2014 - academic.oup.com
Background This study was designed to assess the neuroprotective effect of xenon-induced
delayed postconditioning on spinal cord ischaemia–reperfusion injury (IRI) and to determine …
delayed postconditioning on spinal cord ischaemia–reperfusion injury (IRI) and to determine …
Novel treatments in neuroprotection for aneurysmal subarachnoid hemorrhage
Opinion statement New neuroprotective treatments aimed at preventing or minimizing
“delayed brain injury” are attractive areas of investigation and hold the potential to have …
“delayed brain injury” are attractive areas of investigation and hold the potential to have …
Bradykinin postconditioning ameliorates focal cerebral ischemia in the rat
V Danielisova, M Gottlieb, P Bonova… - Neurochemistry …, 2014 - Elsevier
The goal of this study is to investigate the effects of bradykinin (BR) postconditioning on
cerebral ischemic injury. Transient focal cerebral ischemia was induced in rats by 60 min of …
cerebral ischemic injury. Transient focal cerebral ischemia was induced in rats by 60 min of …
What is the role of erythropoietin in acute myocardial infarct? Bridging the gap between experimental models and clinical trials
F Roubille, F Prunier, S Barrère-Lemaire… - … drugs and therapy, 2013 - Springer
Erythropoietin (EPO) is the main hormone that regulates erythropoiesis. Beyond its well-
known hematopoietic action, EPO has diverse cellular effects in non-hematopoietic tissues …
known hematopoietic action, EPO has diverse cellular effects in non-hematopoietic tissues …
Protective effects of remote ischemic preconditioning in rat hindlimb on ischemia-reperfusion injury★
Y Zhang, X Liu, F Yan, L Min, X Ji… - Neural Regeneration …, 2012 - journals.lww.com
Three cycles of remote ischemic pre-conditioning induced by temporarily occluding the
bilateral femoral arteries (10 minutes) prior to 10 minutes of reperfusion were given once a …
bilateral femoral arteries (10 minutes) prior to 10 minutes of reperfusion were given once a …
Role of mitochondrial function in the protective effects of ischaemic postconditioning on ischaemia/reperfusion cerebral damage
J Liang, H Xu, X Zhang, X Li… - Journal of international …, 2013 - journals.sagepub.com
Objective To investigate the effects of ischaemic postconditioning on brain injury and
mitochondria in focal ischaemia and reperfusion, in rats. Methods Adult male Wistar rats (n …
mitochondria in focal ischaemia and reperfusion, in rats. Methods Adult male Wistar rats (n …
[HTML][HTML] The role of NMDA receptors in the development of brain resistance through pre-and postconditioning
Brain tolerance or resistance can be achieved by interventions before and after injury
through potential toxic agents used in low stimulus or dose. For brain diseases, the …
through potential toxic agents used in low stimulus or dose. For brain diseases, the …
Kainate postconditioning restores LTP in ischemic hippocampal CA1: onset-dependent second pathophysiological stress
Postconditioning can be induced by a broad range of stimuli within minutes to days after an
ischemic cerebral insult. A special form is elicited by pharmacological intervention called …
ischemic cerebral insult. A special form is elicited by pharmacological intervention called …