Persistent infection with human papillomavirus (HPV) initiates~ 5% of all human cancers,
and particularly cervical and oropharyngeal cancers. HPV vaccines prevent HPV infection …

Highlights•Viral infections cause approximately 12% of all human cancers.•Viral cancers
represent rare outcomes of frequent infections.•Oncogenicity of viral proteins reflects their …

There is still controversy in the scientific field about whether certain types of cutaneous
human papillomaviruses (HPVs) are causally involved in the development of non-melanoma …

Double strand breaks (DSBs) are one of the most lethal DNA lesions in cells. The E6 protein
of beta-human papillomavirus (HPV8 E6) impairs two critical DSB repair pathways …

Cutaneous beta genus human papillomaviruses (β-HPVs) are suspected to promote the
development of nonmelanoma skin cancer (NMSC) by destabilizing the host genome …

To replicate, the human papillomaviruses (HPVs) that cause anogenital and oropharyngeal
malignancies must simultaneously activate DNA repair pathways and avoid the cell cycle …

This review focuses on the impact of human papillomavirus (HPV) oncogenes on DNA
repair pathways with a particular focus on how these relationships change as productive …

The beta genus of human papillomaviruses infects cutaneous keratinocytes. Their
replication depends on actively proliferating cells and, thus, they conflict with the cellular …

Infections with cutaneous papillomaviruses have been linked to cutaneous squamous cell
carcinomas that arise in patients who suffer from a rare genetic disorder, epidermodysplasia …

Beta human papillomavirus (beta HPV) infections are common in adults. Certain types of
beta HPVs are associated with nonmelanoma skin cancer (NMSC) in immunocompromised …