Prenatal alcohol exposure (PAE) can reprogram the development of cells and tissues,
resulting in a spectrum of physical and neurobehavioral teratology. PAE immediately …

Diffusion tensor imaging (DTI) has revolutionized our understanding of the neural
underpinnings of alcohol teratogenesis. This technique can detect alterations in white matter …

The neurodevelopmental trajectory in individuals with fetal alcohol spectrum disorders
(FASD) has not been well characterized. We examined age-related differences in the …

Quantitative magnetic resonance imaging (MRI) has revealed abnormalities in brain
volumes, cortical thickness and white matter microstructure in fetal alcohol spectrum …

Prenatal alcohol exposure (PAE) can have immediate and long-lasting toxic and teratogenic
effects on an individual's development and health. As a toxicant, alcohol can lead to a …

Fetal alcohol exposure results in well-characterised neuro-behavioural deficits in offspring,
which form the basis for diagnosing fetal alcohol spectrum disorder. However, there is …

Prenatal alcohol exposure (PAE) can cause central nervous system dysfunction and
widespread structural anomalies as detected by magnetic resonance imaging (MRI). This …

Objectives Mothers who smoke during pregnancy or while their children are small were
common in some populations. Epidemiological studies have tried to detect the effect of …

Despite accumulating evidence from animal models demonstrating that prenatal alcohol
exposure (PAE) results in life-long neuroendocrine dysregulation, very little is known on this …

Background Prenatal alcohol exposure (PAE) can result in physical and neurocognitive
deficits that are collectively termed “fetal alcohol spectrum disorders”(FASD). Although FASD …