The response of immune cells in cardiac injury is divided into three continuous phases:
inflammation, proliferation and maturation. The kinetics of the inflammatory and proliferation …

The dramatic increase in the prevalence of obesity and its strong association with
cardiovascular disease have resulted in unprecedented interest in understanding the effects …

Persistent inflammatory responses participate in the pathogenesis of adverse ventricular
remodeling after myocardial infarction (MI). We hypothesized that regulatory T (Treg) cells …

Cardiac cachexia as a terminal stage of chronic heart failure carries a poor prognosis. The
definition of this clinical syndrome has been a matter of debate in recent years. This review …

Accumulating evidence reveals involvement of T lymphocytes and adaptive immunity in the
chronic inflammation associated with infectious and noninfectious diseases of the heart …

Heart disease is a deadly syndrome affecting millions worldwide. It reflects an unmet clinical
need, and the disease mechanisms are poorly understood. Cardiac fibrosis is central to …

Proinflammatory cytokines are now thought to play a key role in the pathophysiology of
chronic heart failure, driving both symptomatic presentation and disease progression. We …

The polysaccharide extract from Isatidis Radix exhibits potent antiinflammatory and antiviral
activities, but the mechanism of Isatidis Radix polysaccharide (IRP) remains obscure …

Background Excessive immune‐mediated inflammatory reactions play a deleterious role in
postinfarction ventricular remodeling. Interleukin‐37 (IL‐37) emerges as an inhibitor of both …

Over the past 50 years, the understanding of the deteriorative changes involved in the
progression of heart failure (HF), initially described as resulting from changes in salt and …