[HTML][HTML] Intercriteria analysis applied on air pollution influence on morbidity

S Fidanova, P Zhivkov, O Roeva - Mathematics, 2022 - mdpi.com
Human health is reflected in all spheres of life and the economy. One of the main causes of
morbidity and early mortality is polluted air. Ambient air pollution is a serious source of …

Analysis of differentially changed gene expression in EA. hy926 human endothelial cell after exposure of fine particulate matter on the basis of microarray profile

Y Wang, L Xiong, T Wu, T Zhang, L Kong, Y Xue… - Ecotoxicology and …, 2018 - Elsevier
Epidemiological studies have illustrated that PM2. 5 is closely related to cardiovascular
disease (CVD), but underlying toxicological mechanisms are not yet clear. The main …

Prenatal particulate matter exposure and mitochondrial mutational load at the maternal-fetal interface: effect modification by genetic ancestry

KJ Brunst, HHL Hsu, L Zhang, X Zhang, KN Carroll… - Mitochondrion, 2022 - Elsevier
Prenatal ambient particulate matter (PM 2.5) exposure impacts infant development and
alters placental mitochondrial DNA abundance. We investigated whether the timing of PM …

[HTML][HTML] Evidence for the critical role of the PI3K signaling pathway in particulate matter-induced dysregulation of the inflammatory mediators COX-2/PGE2 and the …

C Song, L Liu, J Chen, Y Hu, J Li, B Wang… - Cell Biology and …, 2020 - Springer
Particulate matter (PM) is an environmental pollutant closely associated with human airway
inflammation. However, the molecular mechanisms of PM-related airway inflammation …

Particulate matter 2.5 triggers airway inflammation and bronchial hyperresponsiveness in mice by activating the SIRT2-p65 pathway

M Liu, Z Shi, Y Yin, Y Wang, N Mu, C Li, H Ma… - Frontiers of …, 2021 - Springer
Abstract Exposure to particulate matter 2.5 (PM2. 5) potentially triggers airway inflammation
by activating nuclear factor-κB (NF-κB). Sirtuin 2 (SIRT2) is a key modulator in inflammation …

LncRNA MEG3 involved in NiO NPs-induced pulmonary fibrosis via regulating TGF-β1-mediated PI3K/AKT pathway

H Zhan, X Sun, X Wang, Q Gao, M Yang… - Toxicological …, 2021 - academic.oup.com
Long noncoding RNA maternally expressed gene 3 (MEG3) involves in fibrotic diseases, but
its role in nickel oxide nanoparticles (NiO NPs)-induced pulmonary fibrosis remains unclear …

[HTML][HTML] Pre-admission air pollution exposure prolongs the duration of ventilation in intensive care patients

A De Weerdt, BG Janssen, B Cox, EM Bijnens… - Intensive care …, 2020 - Springer
Purpose Air pollutant exposure constitutes a serious risk factor for the emergence or
aggravation of (existing) pulmonary disease. The impact of pre-intensive care ambient air …

[HTML][HTML] Role of different mechanisms in pro-inflammatory responses triggered by traffic-derived particulate matter in human bronchiolar epithelial cells

M Refsnes, T Skuland, R Jørgensen… - Particle and Fibre …, 2023 - Springer
Background Traffic-derived particles are important contributors to the adverse health effects
of ambient particulate matter (PM). In Nordic countries, mineral particles from road pavement …

Hidden danger: The long-term effect of ultrafine particles on mortality and its sociodemographic disparities in New York State

Q Qi, F Yu, AA Nair, SSS Lau, G Luo, I Mithu… - Journal of Hazardous …, 2024 - Elsevier
Although previous studies have shown increased health risks of particulate matters, few
have evaluated the long-term health impacts of ultrafine particles (UFPs or PM 0.1,≤ 0.1 µm …

Quasi-ultrafine particles promote cell metastasis via HMGB1-mediated cancer cell adhesion

R Gao, N Sang - Environmental Pollution, 2020 - Elsevier
With increasingly severe air pollution, the aggravated health risks of particulate matter,
especially ultrafine particles, are emerging as an urgent and sensitive topic. Considering the …