GGA (geranylgeranylacetone) ameliorates bleomycin-induced lung inflammation and pulmonary fibrosis by inhibiting apoptosis and oxidative stress
R Zhou, C Jin, L Jiao, S Zhang, M Tian, J Liu… - Molecular Biology …, 2023 - Springer
Background Fibrosis is a response to ongoing cellular injury, disruption, and tissue
remodeling, the pathogenesis of which is unknown, and is characterized by extracellular …
remodeling, the pathogenesis of which is unknown, and is characterized by extracellular …
GGA (geranylgeranylacetone) ameliorates bleomycin-induced lung inflammation and pulmonary fibrosis by inhibiting apoptosis and oxidative stress.
R Zhou, C Jin, L Jiao, S Zhang, M Tian, J Liu… - Molecular Biology …, 2023 - europepmc.org
Background Fibrosis is a response to ongoing cellular injury, disruption, and tissue
remodeling, the pathogenesis of which is unknown, and is characterized by extracellular …
remodeling, the pathogenesis of which is unknown, and is characterized by extracellular …
GGA (geranylgeranylacetone) ameliorates bleomycin-induced lung inflammation and pulmonary fibrosis by inhibiting apoptosis and oxidative stress
R Zhou, C Jin, L Jiao, S Zhang… - Molecular biology …, 2023 - pubmed.ncbi.nlm.nih.gov
Background Fibrosis is a response to ongoing cellular injury, disruption, and tissue
remodeling, the pathogenesis of which is unknown, and is characterized by extracellular …
remodeling, the pathogenesis of which is unknown, and is characterized by extracellular …