Aβ [25–35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism: involvement of H2O2, caspase-3, NF-κB, p53 and c-Jun

C Velez-Pardo, GG Ospina, MJ del Rio - Neurotoxicology, 2002 - Elsevier
The Aβ deposition in the neuritic plaques is one of the major neuropathological hallmarks of
the Alzheimer disease (AD). Studies in vitro have demonstrated that the Aβ25–35 fragment …

[25–35] Peptide and Iron Promote Apoptosis in Lymphocytes by an Oxidative Stress Mechanism: Involvement of H 2O 2, Caspase-3, NF-κB, p53 and c-Jun

C Velez-Pardo, G Garcia Ospina… - …, 2002 - ui.adsabs.harvard.edu
The Aβ deposition in the neuritic plaques is one of the major neuropathological hallmarks of
the Alzheimer disease (AD). Studies in vitro have demonstrated that the Aβ 25–35 fragment …

Aβ [2 5-3 5] Peptide and Iron Promote Apoptosis in Lymphocytes by an Oxidative Stress Mechanism: Involvement of H 2 O 2, Caspase-3, NF-κB, p53 and c-Jun

C Velez-Pardo, GG Ospina, MJ del Rio - Neurotoxicology, 2002 - infona.pl
The Aβ deposition in the neuritic plaques is one of the major neuropathological hallmarks of
the Alzheimer disease (AD). Studies in vitro have demonstrated that the Aβ 2 5-3 5 fragment …

Abeta [25-35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism: involvement of H2O2, caspase-3, NF-kappaB, p53 and c-Jun

C Velez-Pardo, GG Ospina, MJ del Rio - Neurotoxicology, 2002 - pubmed.ncbi.nlm.nih.gov
The Abeta deposition in the neuritic plaques is one of the major neuropathological hallmarks
of the Alzheimer disease (AD). Studies in vitro have demonstrated that the Abeta [25-35] …

[PDF][PDF] Ab [25–35] Peptide and Iron Promote Apoptosis in Lymphocytes by an Oxidative Stress Mechanism: Involvement of H2O2, Caspase-3, NF-jB, p53 and c-Jun

C Velez-Pardo, GG Ospina, MJ del Rio - NeuroToxicology, 2002 - researchgate.net
The Ab deposition in the neuritic plaques is one of the major neuropathological hallmarks of
the Alzheimer disease (AD). Studies in vitro have demonstrated that the Aβ25–35 fragment …

Abeta [25-35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism: involvement of H2O2, caspase-3, NF-kappaB, p53 and c-Jun.

C Velez-Pardo, GG Ospina, M Jimenez del Rio - Neurotoxicology, 2002 - europepmc.org
The Abeta deposition in the neuritic plaques is one of the major neuropathological hallmarks
of the Alzheimer disease (AD). Studies in vitro have demonstrated that the Abeta [25-35] …

[引用][C] Aβ [25-35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism: Involvement of H2O2, caspase-3, NF-kB, p53 and c-Jun

C VELEZ-PARDO, G GARCIA OSPINA… - … (Park Forest South), 2002 - pascal-francis.inist.fr
Aβ[25-35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism:
Involvement of H2O2, caspase-3, NF-kB, p53 and c-Jun CNRS Inist Pascal-Francis CNRS Pascal …

[引用][C] Aβ [25-35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism: Involvement of H2O2, caspase-3, NF-kB, p53 and c-Jun

C VELEZ-PARDO, G GARCIA OSPINA… - Neurotoxicology, 2002 - Elsevier