[HTML][HTML] Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1. 5 gating by multiple phosphorylation sites
NM Ashpole, AW Herren, KS Ginsburg… - Journal of Biological …, 2012 - ASBMB
The cardiac Na+ channel Na V 1.5 current (I Na) is critical to cardiac excitability, and altered
I Na gating has been implicated in genetic and acquired arrhythmias. Ca 2+/calmodulin …
I Na gating has been implicated in genetic and acquired arrhythmias. Ca 2+/calmodulin …
Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1. 5 gating by multiple phosphorylation sites
NM Ashpole, AW Herren… - The Journal of …, 2012 - pubmed.ncbi.nlm.nih.gov
The cardiac Na (+) channel Na (V) 1.5 current (I (Na)) is critical to cardiac excitability, and
altered I (Na) gating has been implicated in genetic and acquired arrhythmias. Ca …
altered I (Na) gating has been implicated in genetic and acquired arrhythmias. Ca …
Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1. 5 gating by multiple phosphorylation sites.
NM Ashpole, AW Herren, KS Ginsburg… - The Journal of …, 2012 - europepmc.org
Abstract The cardiac Na+ channel Na V 1.5 current (I Na) is critical to cardiac excitability,
and altered I Na gating has been implicated in genetic and acquired arrhythmias. Ca …
and altered I Na gating has been implicated in genetic and acquired arrhythmias. Ca …
[HTML][HTML] Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV1. 5 Gating by Multiple Phosphorylation Sites
NM Ashpole, AW Herren, KS Ginsburg… - Journal of Biological …, 2012 - Elsevier
The cardiac Na+ channel Na V 1.5 current (I Na) is critical to cardiac excitability, and altered
I Na gating has been implicated in genetic and acquired arrhythmias. Ca 2+/calmodulin …
I Na gating has been implicated in genetic and acquired arrhythmias. Ca 2+/calmodulin …
[引用][C] Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV1. 5 Gating by Multiple Phosphorylation Sites
NM Ashpole, AW Herren, KS Ginsburg… - Journal of Biological …, 2012 - cir.nii.ac.jp
Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium
Channel NaV1.5 Gating by Multiple Phosphorylation Sites | CiNii Research CiNii 国立情報学 …
Channel NaV1.5 Gating by Multiple Phosphorylation Sites | CiNii Research CiNii 国立情報学 …
[HTML][HTML] Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV1. 5 Gating by Multiple Phosphorylation Sites
NM Ashpole, AW Herren, KS Ginsburg… - The Journal of …, 2012 - ncbi.nlm.nih.gov
Background: CaMKII is up-regulated in heart failure and modulates Na+ current (I Na), yet
the mechanism is unclear. Result: CaMKII phosphorylates several sites in the first …
the mechanism is unclear. Result: CaMKII phosphorylates several sites in the first …
[PDF][PDF] Ca 2/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV 1.5 Gating by Multiple Phosphorylation Sites
NM Ashpole, AW Herren, KS Ginsburg… - THE JOURNAL OF …, 2012 - researchgate.net
Background: CaMKII is up-regulated in heart failure and modulates Na current (INa), yet the
mechanism is unclear. Result: CaMKII phosphorylates several sites in the first intracellular …
mechanism is unclear. Result: CaMKII phosphorylates several sites in the first intracellular …
Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1. 5 gating by multiple phosphorylation sites.
NM Ashpole, AW Herren, KS Ginsburg… - The Journal of …, 2012 - europepmc.org
Abstract The cardiac Na+ channel Na V 1.5 current (I Na) is critical to cardiac excitability,
and altered I Na gating has been implicated in genetic and acquired arrhythmias. Ca …
and altered I Na gating has been implicated in genetic and acquired arrhythmias. Ca …