NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker, E Rampanelli… - Journal of Experimental …, 2017 - rupress.org
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

[HTML][HTML] NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker, E Rampanelli… - Journal of Experimental …, 2017 - rupress.org
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker, E Rampanelli… - Journal of Experimental …, 2017 - elibrary.ru
G. Stokman is supported by a grant from the Dutch Kidney Foundation (13A3D301). PJ
Bakker, L. Kors, and E. Rampanelli are supported by a VIDI grant from the Netherlands …

NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker, E Rampanelli… - Journal of Experimental …, 2017 - cir.nii.ac.jp
抄録< jats: p> Mitochondrial dysfunction is the most prominent source of oxidative stress in
acute and chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity.

G Stokman, L Kors, PJ Bakker… - The Journal of …, 2017 - europepmc.org
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

[PDF][PDF] NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker, E Rampanelli… - J. Exp. Med, 2017 - academia.edu
TLRs and NOD-like receptors (NLRs; also known as nucleotide-binding, lots of leucine-rich
repeats-containing protein members) are important regulators of innate immunity during …

[HTML][HTML] NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker… - The Journal of …, 2017 - ncbi.nlm.nih.gov
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker… - The Journal of …, 2017 - pubmed.ncbi.nlm.nih.gov
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity

G Stokman, L Kors, PJ Bakker… - Journal of …, 2017 - researchinformation.amsterdamumc …
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …

NLRX1 dampens oxidative stress and apoptosis in tissue injury via control of mitochondrial activity.

G Stokman, L Kors, PJ Bakker… - The Journal of …, 2017 - europepmc.org
Mitochondrial dysfunction is the most prominent source of oxidative stress in acute and
chronic kidney disease. NLRX1 is a receptor of the innate immune system that is …