Leishmanial lipid affords protection against oxidative stress induced hepatic injury by regulating inflammatory mediators and confining apoptosis progress

N Chatterjee, S Das, D Bose, S Banerjee, T Jha… - Toxicology Letters, 2015 - Elsevier
Persistence of liver injury alters the internal milieu, promotes deregulation of inflammatory
factors, and leads to dysplastic lesions like fibrosis, cirrhosis to hepatocellular carcinoma …
被引用次数：18 相关文章

Leishmanial lipid affords protection against oxidative stress induced hepatic injury by regulating inflammatory mediators and confining apoptosis progress.

N Chatterjee, S Das, D Bose, S Banerjee, T Jha… - Toxicology …, 2014 - europepmc.org
Persistence of liver injury alters the internal milieu, promotes deregulation of inflammatory
factors, and leads to dysplastic lesions like fibrosis, cirrhosis to hepatocellular carcinoma …

Leishmanial lipid affords protection against oxidative stress induced hepatic injury by regulating inflammatory mediators and confining apoptosis progress

N Chatterjee, S Das, D Bose, S Banerjee, T Jha… - Toxicology Letters, 2015 - infona.pl
Persistence of liver injury alters the internal milieu, promotes deregulation of inflammatory
factors, and leads to dysplastic lesions like fibrosis, cirrhosis to hepatocellular carcinoma …

Leishmanial lipid affords protection against oxidative stress induced hepatic injury by regulating inflammatory mediators and confining apoptosis progress

N Chatterjee, S Das, D Bose, S Banerjee… - Toxicology …, 2015 - pubmed.ncbi.nlm.nih.gov
Persistence of liver injury alters the internal milieu, promotes deregulation of inflammatory
factors, and leads to dysplastic lesions like fibrosis, cirrhosis to hepatocellular carcinoma …