[HTML][HTML] System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations

B Xu, E Holmes, PC Guest, M Karayiorgou… - Molecular …, 2017 - nature.com
Abstract Deletions on chromosome 22q11. 2 are a strong genetic risk factor for development
of schizophrenia and cognitive dysfunction. We employed shotgun liquid chromatography …

[PDF][PDF] System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations

H Wesseling, B Xu, EJ Want, E Holmes, PC Guest… - Molecular …, 2017 - core.ac.uk
Hemizygous deletions in chromosome 22q11. 2 occur predominately de novo and cause a
deletion syndrome (22q11. 2DS), characterized by a broad spectrum of physical and mental …

System-based proteomic and metabonomic analysis of the Df(16)A+/- mouse identifies potential miR-185 targets and molecular pathway alterations.

H Wesseling, B Xu, EJ Want, E Holmes… - Molecular …, 2016 - europepmc.org
Abstract Deletions on chromosome 22q11. 2 are a strong genetic risk factor for development
of schizophrenia and cognitive dysfunction. We employed shotgun liquid chromatography …

System-based proteomic and metabonomic analysis of the Df (16) A+/-mouse identifies potential miR-185 targets and molecular pathway alterations

H Wesseling, B Xu, EJ Want, E Holmes, P Guest… - Molecular …, 2017 - repub.eur.nl
Deletions on chromosome 22q11. 2 are a strong genetic risk factor for development of
schizophrenia and cognitive dysfunction. We employed shotgun liquid chromatography …

System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations.

H Wesseling, B Xu, EJ Want, E Holmes… - Molecular …, 2017 - psycnet.apa.org
Abstract Deletions on chromosome 22q11. 2 are a strong genetic risk factor for development
of schizophrenia and cognitive dysfunction. We employed shotgun liquid chromatography …

[引用][C] System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations

H Wesseling, B Xu, EJ Want, E Holmes, PC Guest… - Molecular …, 2016 - cir.nii.ac.jp
System-based proteomic and metabonomic analysis of the Df(16)A+/− mouse identifies
potential miR-185 targets and molecular pathway alterations | CiNii Research CiNii 国立情報学 …

[PDF][PDF] System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations

Molecular Psychiatry, 2016 - researchgate.net
Hemizygous deletions in chromosome 22q11. 2 occur predominately de novo and cause a
deletion syndrome (22q11. 2DS), characterized by a broad spectrum of physical and mental …

[PDF][PDF] System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations

H Wesseling, B Xu, EJ Want, E Holmes, PC Guest… - Molecular …, 2017 - pure.eur.nl
Hemizygous deletions in chromosome 22q11. 2 occur predominately de novo and cause a
deletion syndrome (22q11. 2DS), characterized by a broad spectrum of physical and mental …

System-based proteomic and metabonomic analysis of the Df (16) A+/-mouse identifies potential miR-185 targets and molecular pathway alterations

H Wesseling, B Xu, EJ Want, E Holmes… - Molecular …, 2017 - pubmed.ncbi.nlm.nih.gov
Deletions on chromosome 22q11. 2 are a strong genetic risk factor for development of
schizophrenia and cognitive dysfunction. We employed shotgun liquid chromatography …

[HTML][HTML] System-based proteomic and metabonomic analysis of the Df (16) A+/− mouse identifies potential miR-185 targets and molecular pathway alterations

H Wesseling, B Xu, EJ Want, E Holmes… - Molecular …, 2017 - ncbi.nlm.nih.gov
Abstract Deletions on chromosome 22q11. 2 are a strong genetic risk factor for development
of schizophrenia and cognitive dysfunction. We employed shotgun liquid chromatography …