A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice

X Cai, H Liu, B Feng, M Yu, Y He, H Liu, C Liang… - Nature …, 2022 - nature.com
Midbrain dopamine (DA) and serotonin (5-HT) neurons regulate motivated behaviors,
including feeding, but less is known about how these circuits may interact. In this study, we …

A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice.

X Cai, H Liu, B Feng, M Yu, Y He, C Liang… - Nature …, 2022 - europepmc.org
Midbrain dopamine (DA) and serotonin (5-HT) neurons regulate motivated behaviors,
including feeding, but less is known about how these circuits may interact. In this study, we …

[PDF][PDF] A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice

X Cai, H Liu, B Feng, M Yu, Y He, H Liu… - Nature …, 2022 - drive.google.com
Midbrain dopamine (DA) and serotonin (5-HT) neurons regulate motivated behaviors,
including feeding, but less is known about how these circuits may interact. In this study, we …

A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice

X CAI - 2022 - ars.usda.gov
This study investigated the interaction between dopamine (DA) and serotonin (5-HT)
neurons in the brain and how they regulate feeding behaviors. The researchers found that …

A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice

X Cai, H Liu, B Feng, M Yu, Y He, H Liu… - Nature …, 2022 - pubmed.ncbi.nlm.nih.gov
Midbrain dopamine (DA) and serotonin (5-HT) neurons regulate motivated behaviors,
including feeding, but less is known about how these circuits may interact. In this study, we …

[HTML][HTML] A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice

X Cai, H Liu, B Feng, M Yu, Y He, H Liu… - Nature …, 2022 - ncbi.nlm.nih.gov
Midbrain dopamine (DA) and serotonin (5-HT) neurons regulate motivated behaviors,
including feeding, but less is known about how these circuits may interact. In this study, we …