Proteomics reveals Rictor as a noncanonical TGF-β signaling target during aneurysm progression in Marfan mice

SJ Parker, A Stotland, E MacFarlane… - American Journal …, 2018 - journals.physiology.org
The objective of the present study was to 1) analyze the ascending aortic proteome within a
mouse model of Marfan syndrome (MFS; Fbn1 C1041G/+) at early and late stages of …

Proteomics Reveals Rictor as a Non-Canonical TGFB Signaling Target During Aneurysm 1 Progression in Marfan Mice 2

SJ Parker - journals.physiology.org
36 We present the most comprehensive quantitative analysis of the ascending aortic
aneurysm 37 proteome in Marfan Syndrome to date resulting in novel and potentially wide …

[PDF][PDF] Proteomics reveals Rictor as a noncanonical TGF-signaling target during aneurysm progression in Marfan mice

SJ Parker, A Stotland, E MacFarlane… - Am J Physiol Heart …, 2018 - scholar.archive.org
Aneurysm, or dilation of the proximal thoracic aorta, is a highly penetrant and life-threatening
phenotype in individuals with Marfan syndrome (MFS), a connective tissue disorder caused …

Proteomics reveals Rictor as a noncanonical TGF-β signaling target during aneurysm progression in Marfan mice.

SJ Parker, A Stotland, E MacFarlane… - American Journal of …, 2018 - europepmc.org
The objective of the present study was to 1) analyze the ascending aortic proteome within a
mouse model of Marfan syndrome (MFS; Fbn1 C1041G/+) at early and late stages of …

Proteomics reveals Rictor as a noncanonical TGF-β signaling target during aneurysm progression in Marfan mice

SJ Parker, A Stotland, E MacFarlane… - American journal …, 2018 - pubmed.ncbi.nlm.nih.gov
The objective of the present study was to 1) analyze the ascending aortic proteome within a
mouse model of Marfan syndrome (MFS; Fbn1 C1041G/+) at early and late stages of …

Proteomics reveals Rictor as a noncanonical TGF-β signaling target during aneurysm progression in Marfan mice.

SJ Parker, A Stotland, E MacFarlane… - American Journal of …, 2018 - europepmc.org
The objective of the present study was to 1) analyze the ascending aortic proteome within a
mouse model of Marfan syndrome (MFS; Fbn1 C1041G/+) at early and late stages of …