SIRT1-dependent PGC-1α deacetylation by SRT1720 rescues progression of atherosclerosis by enhancing mitochondrial function
JY Sung, SG Kim, YJ Kang, SY Park… - Biochimica et Biophysica …, 2024 - Elsevier
Vascular smooth muscle cell (VSMC) senescence promotes atherosclerosis via lipid-
mediated mitochondrial dysfunction and oxidative stress. However, the mechanisms of …
mediated mitochondrial dysfunction and oxidative stress. However, the mechanisms of …
SIRT1-dependent PGC-1α deacetylation by SRT1720 rescues progression of atherosclerosis by enhancing mitochondrial function
JY Sung, SG Kim, YJ Kang… - … et biophysica acta …, 2024 - pubmed.ncbi.nlm.nih.gov
Vascular smooth muscle cell (VSMC) senescence promotes atherosclerosis via lipid-
mediated mitochondrial dysfunction and oxidative stress. However, the mechanisms of …
mediated mitochondrial dysfunction and oxidative stress. However, the mechanisms of …
SIRT1-dependent PGC-1α deacetylation by SRT1720 rescues progression of atherosclerosis by enhancing mitochondrial function.
JY Sung, SG Kim, YJ Kang, SY Park… - … et Biophysica acta …, 2024 - europepmc.org
Vascular smooth muscle cell (VSMC) senescence promotes atherosclerosis via lipid-
mediated mitochondrial dysfunction and oxidative stress. However, the mechanisms of …
mediated mitochondrial dysfunction and oxidative stress. However, the mechanisms of …