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Cellular senescence is increased in airway smooth muscle cells of elderly persons with asthma

A Aghali, L Khalfaoui, AB Lagnado… - … of Physiology-Lung …, 2022 - journals.physiology.org
Senescent cells can drive age-related tissue dysfunction partially via a senescence-
associated secretory phenotype (SASP) involving proinflammatory and profibrotic factors …
被引用次数：18 相关文章

Cellular senescence is increased in airway smooth muscle cells of elderly persons with asthma

A Aghali, L Khalfaoui, AB Lagnado… - … Lung cellular and …, 2022 - mayoclinic.elsevierpure.com
Senescent cells can drive age-related tissue dysfunction partially via a senescence-
associated secretory phenotype (SASP) involving proinflammatory and profibrotic factors …

Cellular senescence is increased in airway smooth muscle cells of elderly persons with asthma.

A Aghali, L Khalfaoui, AB Lagnado… - American Journal of …, 2022 - europepmc.org
Cellular senescence is increased in airway smooth muscle cells of elderly persons with asthma.
- Abstract - Europe PMC Sign in | Create an account https://orcid.org Europe PMC Menu About …
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[HTML][HTML] Senescence in the Lung: Cellular senescence is increased in airway smooth muscle cells of elderly persons with asthma

A Aghali, L Khalfaoui, AB Lagnado… - American Journal of …, 2022 - ncbi.nlm.nih.gov
Senescent cells can drive age-related tissue dysfunction partially via a senescence-
associated secretory phenotype (SASP) involving proinflammatory and profibrotic factors …

Cellular senescence is increased in airway smooth muscle cells of elderly persons with asthma

A Aghali, L Khalfaoui, AB Lagnado… - … . Lung cellular and …, 2022 - pubmed.ncbi.nlm.nih.gov
Senescent cells can drive age-related tissue dysfunction partially via a senescence-
associated secretory phenotype (SASP) involving proinflammatory and profibrotic factors …

Cellular Senescence is Increased in Airway Smooth Muscle Cells of Elderly Asthmatics.

A Aghali, L Khalfaoui, AB Lagnado, L Drake… - American Journal of …, 2022 - europepmc.org
Senescent cells can drive age-related tissue dysfunction partially via a senescence-
associated secretory phenotype (SASP) involving pro-inflammatory and pro-fibrotic factors …