[HTML][HTML] Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular …

KMA Dreijerink, E Ozyerli-Goknar, S Koidl… - Epigenetics & …, 2022 - Springer
Background Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular pathogenicity.

KMA Dreijerink, E Ozyerli-Goknar, S Koidl… - Epigenetics & …, 2022 - europepmc.org
Background Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

[PDF][PDF] Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular …

KMA Dreijerink, E Ozyerli-Goknar, S Koidl… - 2022 - academia.edu
Background: Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

[HTML][HTML] Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular …

KMA Dreijerink, E Ozyerli-Goknar, S Koidl… - Epigenetics & …, 2022 - ncbi.nlm.nih.gov
Background Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular pathogenicity

KMA Dreijerink… - Epigenetics & …, 2022 - researchinformation.amsterdamumc …
BACKGROUND: Loss-of-function mutations of the multiple endocrine neoplasia type 1
(MEN1) gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular …

KMA Dreijerink, E Ozyerli-Goknar… - Epigenetics & …, 2022 - search.proquest.com
Background Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular pathogenicity.

K Dreijerink, E Ozyerli-Goknar, S Koidl… - Epigenetics & …, 2022 - search.ebscohost.com
Background: Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …

[PDF][PDF] Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular …

KMA Dreijerink, E Ozyerli-Goknar, S Koidl… - scholar.archive.org
Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1) gene are
causal to the MEN1 tumor syndrome, but they are also commonly found in sporadic …

Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular pathogenicity

KMA Dreijerink, E Ozyerli-Goknar, S Koidl… - bioRxiv, 2022 - biorxiv.org
Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1) gene are
causal to the MEN1 tumor syndrome, but they are also commonly found in sporadic …

Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular pathogenicity

KMA Dreijerink, E Ozyerli-Goknar… - Epigenetics & …, 2022 - pubmed.ncbi.nlm.nih.gov
Background Loss-of-function mutations of the multiple endocrine neoplasia type 1 (MEN1)
gene are causal to the MEN1 tumor syndrome, but they are also commonly found in …