A mitochondrial contribution to anti-inflammatory shear stress signaling in vascular endothelial cells

BG Coon, S Timalsina, M Astone, ZW Zhuang… - Journal of Cell …, 2022 - rupress.org
Atherosclerosis, the major cause of myocardial infarction and stroke, results from converging
inflammatory, metabolic, and biomechanical factors. Arterial lesions form at sites of low and …

[HTML][HTML] A mitochondrial contribution to anti-inflammatory shear stress signaling in vascular endothelial cells

BG Coon, S Timalsina, M Astone… - The Journal of Cell …, 2022 - ncbi.nlm.nih.gov
Atherosclerosis, the major cause of myocardial infarction and stroke, results from converging
inflammatory, metabolic, and biomechanical factors. Arterial lesions form at sites of low and …

A mitochondrial contribution to anti-inflammatory shear stress signaling in vascular endothelial cells

BG Coon, S Timalsina, M Astone… - The Journal of cell …, 2022 - pubmed.ncbi.nlm.nih.gov
Atherosclerosis, the major cause of myocardial infarction and stroke, results from converging
inflammatory, metabolic, and biomechanical factors. Arterial lesions form at sites of low and …

A mitochondrial contribution to anti-inflammatory shear stress signaling in vascular endothelial cells.

BG Coon, S Timalsina, M Astone… - The Journal of Cell …, 2022 - europepmc.org
Atherosclerosis, the major cause of myocardial infarction and stroke, results from converging
inflammatory, metabolic, and biomechanical factors. Arterial lesions form at sites of low and …

A mitochondrial contribution to anti-inflammatory shear stress signaling in vascular endothelial cells

BG Coon, S Timalsina, M Astone… - THE JOURNAL OF …, 2022 - research.unipd.it
Atherosclerosis, the major cause of myocardial infarction and stroke, results from converging
inflammatory, metabolic, and biomechanical factors. Arterial lesions form at sites of low and …