Endogenous tryptophan metabolite 5-Methoxytryptophan inhibits pulmonary fibrosis by downregulating the TGF-β/SMAD3 and PI3K/AKT signaling pathway

L Fang, H Chen, R Kong, J Que - Life sciences, 2020 - Elsevier
Pulmonary fibrosis is the end stage of many interstitial lung diseases, characterized by the
deposition of excess extracellular matrix (ECM), destruction of normal alveolar structure, and …

Endogenous tryptophan metabolite 5-Methoxytryptophan inhibits pulmonary fibrosis by downregulating the TGF-β/SMAD3 and PI3K/AKT signaling pathway.

L Fang, H Chen, R Kong, J Que - Life Sciences, 2020 - europepmc.org
Pulmonary fibrosis is the end stage of many interstitial lung diseases, characterized by the
deposition of excess extracellular matrix (ECM), destruction of normal alveolar structure, and …

Endogenous tryptophan metabolite 5-Methoxytryptophan inhibits pulmonary fibrosis by downregulating the TGF-β/SMAD3 and PI3K/AKT signaling pathway

L Fang, H Chen, R Kong, J Que - Life sciences, 2020 - pubmed.ncbi.nlm.nih.gov
Pulmonary fibrosis is the end stage of many interstitial lung diseases, characterized by the
deposition of excess extracellular matrix (ECM), destruction of normal alveolar structure, and …

[引用][C] Endogenous tryptophan metabolite 5-Methoxytryptophan inhibits pulmonary fibrosis by downregulating the TGF-β/SMAD3 and PI3K/AKT signaling pathway

L Fang, H Chen, R Kong, J Que - 2020