GBT 440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half‐life in a murine model of sickle cell disease
D Oksenberg, K Dufu, MP Patel… - British journal of …, 2016 - Wiley Online Library
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
GBT440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half-life in a murine model of sickle cell disease.
D Oksenberg, K Dufu, MP Patel… - British Journal of …, 2016 - search.ebscohost.com
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
GBT 440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half‐life in a murine model of sickle cell disease
D Oksenberg, K Dufu, MP Patel, C Chuang, Z Li… - British Journal of …, 2016 - infona.pl
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
GBT440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half-life in a murine model of sickle cell disease
D Oksenberg, K Dufu, MP Patel… - British Journal of …, 2016 - einstein.elsevierpure.com
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
GBT440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half‐life in a murine model of sickle cell disease
D Oksenberg, K Dufu, MP Patel, C Chuang… - British Journal of …, 2016 - cir.nii.ac.jp
抄録< jats: title> Summary</jats: title>< jats: p> A major driver of the pathophysiology of
sickle cell disease (< jats: styled-content style=" fixed-case"> SCD</jats: styled-content>) is …
sickle cell disease (< jats: styled-content style=" fixed-case"> SCD</jats: styled-content>) is …
GBT440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half-life in a murine model of sickle cell disease
D Oksenberg, K Dufu, MP Patel… - British journal of …, 2016 - pubmed.ncbi.nlm.nih.gov
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
GBT440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half-life in a murine model of sickle cell disease.
D Oksenberg, K Dufu, MP Patel, C Chuang… - British Journal of …, 2016 - europepmc.org
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
GBT 440 increases haemoglobin oxygen affinity, reduces sickling and prolongs RBC half‐life in a murine model of sickle cell disease
D Oksenberg, K Dufu, MP Patel, C Chuang, Z Li… - British Journal of …, 2016 - infona.pl
A major driver of the pathophysiology of sickle cell disease (SCD) is polymerization of
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …
deoxygenated haemoglobin S (HbS), which leads to sickling and destruction of red blood …