[HTML][HTML] Exercise training alleviates cardiac fibrosis through increasing fibroblast growth factor 21 and regulating TGF-β1-Smad2/3-MMP2/9 signaling in mice with …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu, M Cai… - International Journal of …, 2021 - mdpi.com
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice with …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu… - International …, 2021 - search.ebscohost.com
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice with …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu, M Cai, Z Tian - agris.fao.org
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
[PDF][PDF] Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice with …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu… - Int. J. Mol …, 2021 - pdfs.semanticscholar.org
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice with …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu… - International Journal of …, 2021 - europepmc.org
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice with …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu… - … Journal of Molecular …, 2021 - search.proquest.com
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice with …
Y Ma, Y Kuang, W Bo, Q Liang… - International …, 2021 - pubmed.ncbi.nlm.nih.gov
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
[HTML][HTML] Exercise Training Alleviates Cardiac Fibrosis through Increasing Fibroblast Growth Factor 21 and Regulating TGF-β1-Smad2/3-MMP2/9 Signaling in Mice …
Y Ma, Y Kuang, W Bo, Q Liang, W Zhu… - International Journal of …, 2021 - ncbi.nlm.nih.gov
Exercise training has been reported to alleviate cardiac fibrosis and ameliorate heart
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …
dysfunction after myocardial infarction (MI), but the molecular mechanism is still not fully …