[HTML][HTML] An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (AT)

AJ Yeo, GN Subramanian, KL Chong, M Gatei… - Molecular …, 2021 - Elsevier
Background ATM, the protein defective in the human genetic disorder, ataxia-telangiectasia
(AT) plays a central role in response to DNA double-strand breaks (DSBs) and in protecting …

[PDF][PDF] An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (AT)

AJ Yeo, GN Subramanian, KL Chong, M Gatei… - 2021 - researchgate.net
Background: ATM, the protein defective in the human genetic disorder, ataxia-telangiectasia
(AT) plays a central role in response to DNA double-strand breaks (DSBs) and in protecting …

[引用][C] An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (AT)

AJ Yeo, GN Subramanian, KL Chong… - Molecular …, 2021 - espace.library.uq.edu.au
ATM, the protein defective in the human genetic disorder, ataxia telangiectasia (AT) plays a
central role in the response to DNA double strand breaks (DSBs) and in protecting the cell …

An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (AT)

AJ Yeo, GN Subramanian, KL Chong… - Molecular …, 2021 - pubmed.ncbi.nlm.nih.gov
Background ATM, the protein defective in the human genetic disorder, ataxia-telangiectasia
(AT) plays a central role in response to DNA double-strand breaks (DSBs) and in protecting …

[HTML][HTML] An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (AT)

AJ Yeo, GN Subramanian, KL Chong, M Gatei… - Molecular …, 2021 - ncbi.nlm.nih.gov
Background ATM, the protein defective in the human genetic disorder, ataxia-telangiectasia
(AT) plays a central role in response to DNA double-strand breaks (DSBs) and in protecting …

An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (AT).

AJ Yeo, GN Subramanian, KL Chong, M Gatei… - Molecular …, 2021 - europepmc.org
Background ATM, the protein defective in the human genetic disorder, ataxia-telangiectasia
(AT) plays a central role in response to DNA double-strand breaks (DSBs) and in protecting …