[HTML][HTML] The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis
Intestinal goblet cells are potentially key players in controlling susceptibility to ulcerative
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis
V Morampudi, U Dalwadi, G Bhinder… - Mucosal …, 2016 - pubmed.ncbi.nlm.nih.gov
Intestinal goblet cells are potentially key players in controlling susceptibility to ulcerative
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
[HTML][HTML] The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis
V Morampudi, U Dalwadi, G Bhinder… - Mucosal …, 2016 - mucosalimmunology.org
Intestinal goblet cells are potentially key players in controlling susceptibility to ulcerative
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
[HTML][HTML] The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis
V Morampudi, U Dalwadi, G Bhinder, HP Sham… - Mucosal …, 2016 - nature.com
Intestinal goblet cells are potentially key players in controlling susceptibility to ulcerative
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
[引用][C] The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis
V Morampudi, U Dalwadi, G Bhinder, HP Sham… - Mucosal …, 2016 - cir.nii.ac.jp
The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice
by promoting commensal microbial dysbiosis | CiNii Research CiNii 国立情報学研究所 学術 …
by promoting commensal microbial dysbiosis | CiNii Research CiNii 国立情報学研究所 学術 …
The goblet cell-derived mediator RELM-β drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis.
V Morampudi, U Dalwadi, G Bhinder, HP Sham… - Mucosal …, 2016 - europepmc.org
Intestinal goblet cells are potentially key players in controlling susceptibility to ulcerative
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
The goblet cell-derived mediator RELM-[beta] drives spontaneous colitis in Muc2-deficient mice by promoting commensal microbial dysbiosis
V Morampudi, U Dalwadi, G Bhinder… - Mucosal …, 2016 - search.proquest.com
Intestinal goblet cells are potentially key players in controlling susceptibility to ulcerative
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …
colitis (UC). Although impaired mucin (Muc2) production by goblet cells increases microbial …