[HTML][HTML] N-methyl-D-aspartate receptor-and calpain-mediated proteolytic cleavage of K+-Cl− cotransporter-2 impairs spinal chloride homeostasis in neuropathic pain

HY Zhou, SR Chen, HS Byun, H Chen, L Li… - Journal of Biological …, 2012 - ASBMB
Loss of synaptic inhibition by γ-aminobutyric acid and glycine due to potassium chloride
cotransporter-2 (KCC2) down-regulation in the spinal cord is a critical mechanism of …

[HTML][HTML] N-Methyl-d-aspartate Receptor-and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic …

HY Zhou, SR Chen, HS Byun, H Chen, L Li… - Journal of Biological …, 2012 - Elsevier
Loss of synaptic inhibition by γ-aminobutyric acid and glycine due to potassium chloride
cotransporter-2 (KCC2) down-regulation in the spinal cord is a critical mechanism of …

[引用][C] N-Methyl-d-aspartate Receptor-and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic …

HY Zhou, SR Chen, HS Byun, H Chen, L Li… - Journal of Biological …, 2012 - cir.nii.ac.jp
N-Methyl-d-aspartate Receptor- and Calpain-mediated Proteolytic Cleavage of K+-Cl−
Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic Pain | CiNii Research …

N-methyl-D-aspartate receptor-and calpain-mediated proteolytic cleavage of K+-Cl-cotransporter-2 impairs spinal chloride homeostasis in neuropathic pain

HY Zhou, SR Chen, HS Byun… - The Journal of …, 2012 - pubmed.ncbi.nlm.nih.gov
Loss of synaptic inhibition by γ-aminobutyric acid and glycine due to potassium chloride
cotransporter-2 (KCC2) down-regulation in the spinal cord is a critical mechanism of …

N-methyl-D-aspartate receptor-and calpain-mediated proteolytic cleavage of K+-Cl-cotransporter-2 impairs spinal chloride homeostasis in neuropathic pain.

HY Zhou, SR Chen, HS Byun, H Chen, L Li… - The Journal of …, 2012 - europepmc.org
Loss of synaptic inhibition by γ-aminobutyric acid and glycine due to potassium chloride
cotransporter-2 (KCC2) down-regulation in the spinal cord is a critical mechanism of …

[HTML][HTML] N-Methyl-d-aspartate Receptor-and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic …

HY Zhou, SR Chen, HS Byun, H Chen, L Li… - The Journal of …, 2012 - ncbi.nlm.nih.gov
Background: Reduced K+-Cl− cotransporter-2 (KCC2) function in spinal cords contributes to
diminished synaptic inhibition and neuropathic pain development. Results: Nerve injury …

N-methyl-D-aspartate receptor-and calpain-mediated proteolytic cleavage of K+-Cl-cotransporter-2 impairs spinal chloride homeostasis in neuropathic pain

HY Zhou, SR Chen, HS Byun… - Journal of …, 2012 - mdanderson.elsevierpure.com
Loss of synaptic inhibition by γ-aminobutyric acid and glycine due to potassium chloride
cotransporter-2 (KCC2) downregulation in the spinal cord is a critical mechanism of synaptic …

N-methyl-D-aspartate receptor-and calpain-mediated proteolytic cleavage of K+-Cl-cotransporter-2 impairs spinal chloride homeostasis in neuropathic pain.

HY Zhou, SR Chen, HS Byun, H Chen, L Li… - The Journal of …, 2012 - europepmc.org
Loss of synaptic inhibition by γ-aminobutyric acid and glycine due to potassium chloride
cotransporter-2 (KCC2) down-regulation in the spinal cord is a critical mechanism of …