The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin
AV Birk, S Liu, YI Soong, W Mills, P Singh… - Journal of the …, 2013 - journals.lww.com
Ischemia causes AKI as a result of ATP depletion, and rapid recovery of ATP on reperfusion
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
[HTML][HTML] The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by Interacting with Cardiolipin
AV Birk, S Liu, Y Soong, W Mills, P Singh… - Journal of the …, 2013 - ncbi.nlm.nih.gov
Ischemia causes AKI as a result of ATP depletion, and rapid recovery of ATP on reperfusion
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
[引用][C] The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by Interacting with Cardiolipin
AV BIRK, S LIU, YI SOONG, W MILLS… - Journal of the …, 2013 - pascal-francis.inist.fr
The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by
Interacting with Cardiolipin CNRS Inist Pascal-Francis CNRS Pascal and Francis …
Interacting with Cardiolipin CNRS Inist Pascal-Francis CNRS Pascal and Francis …
[引用][C] The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by Interacting with Cardiolipin
AV Birk, S Liu, Y Soong, W Mills, P Singh… - Journal of the American …, 2013 - cir.nii.ac.jp
The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by
Interacting with Cardiolipin | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] …
Interacting with Cardiolipin | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] …
[PDF][PDF] The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by Interacting with Cardiolipin
AV Birk, S Liu, Y Soong, W Mills, P Singh… - J Am Soc …, 2013 - researchgate.net
Ischemia causes AKI as a result of ATP depletion, and rapid recovery of ATP on reperfusion
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin
AV Birk, S Liu, Y Soong, W Mills… - Journal of the …, 2013 - pubmed.ncbi.nlm.nih.gov
Ischemia causes AKI as a result of ATP depletion, and rapid recovery of ATP on reperfusion
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin.
AV Birk, S Liu, Y Soong, W Mills, P Singh… - Journal of the …, 2013 - europepmc.org
Ischemia causes AKI as a result of ATP depletion, and rapid recovery of ATP on reperfusion
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin.
AV Birk, S Liu, Y Soong, W Mills, P Singh… - Journal of the …, 2013 - europepmc.org
Ischemia causes AKI as a result of ATP depletion, and rapid recovery of ATP on reperfusion
is important to minimize tissue damage. ATP recovery is often delayed, however, because …
is important to minimize tissue damage. ATP recovery is often delayed, however, because …