Nicotine alters cognitive functions in animals and humans most likely by modification of
brain plasticity. In the human brain, it alters plasticity induced by transcranial direct current …

Nicotine modulates neuroplasticity and improves cognitive functions in animals and
humans. In the brain of smoking individuals, calcium-dependent plasticity induced by non …

Nicotine improves cognitive performance and modulates neuroplasticity in brain networks.
The neurophysiological mechanisms underlying nicotine-induced behavioral changes have …

The impact of nicotine (NIC) on plasticity is thought to be primarily determined via calcium
channel properties of nicotinic receptor subtypes, and glutamatergic plasticity is likewise …

Key points Nicotine (NIC) modulates cognition and memory function by targeting the
nicotinic ACh receptor and releasing different transmitter systems postsynaptically. With both …

In various studies nicotine has shown to alter cognitive functions in non-smoking subjects.
The physiological basis for these effects might be nicotine-generated modulation of cortical …

Nicotine improves cognitive functions by modulating neuroplasticity and cortical excitability
in nonsmoking subjects. As shown recently, the positive effect of nicotine on cognition might …

Nicotine has been shown to modulate neuroplasticity, cognition, and learning processes in
smokers and non-smokers. A possible mechanism for its effect on learning and memory …

Amphetamine, a catecholaminergic re-uptake-blocker, is able to improve neuroplastic
mechanisms in humans. However, so far not much is known about the underlying …

Cholinergic neuromodulation is pivotal for arousal, attention, and cognitive processes. Loss
or dysregulation of cholinergic inputs leads to cognitive impairments like those manifested in …