Enhanced neovasculogenesis, especially vasculogenic mimicry (VM), contributes to the
development of triple-negative breast cancer (TNBC). Breast tumor-initiating cells (BTICs) …

Vasculogenic mimicry (VM) is a nonangiogenesis-dependent pathway that promotes tumor
growth and disease progression. Nodal signaling has several vital roles in both embryo …

Triple-negative breast cancer (TNBC) is highly aggressive with limited available treatments.
Stromal cells in the tumor microenvironment (TME) are crucial in TNBC progression; …

In solid tumors, vasculogenic mimicry (VM) is the formation of vascular structures by cancer
cells, allowing to generate a channel-network able to transport blood and tumor cells. While …

Agents that target angiogenesis have shown limited efficacy for human triple-negative breast
cancer (TNBC) in clinical trials. Along with endothelium-dependent vessels, there is also …

Objective Vasculogenic mimicry (VM) channels that are lined by tumor cells are a functional
blood supply in malignant tumors. However, the role of VM-initiating cells remains poorly …

Triple-negative breast cancer (TNBC) is associated with epithelial-mesenchymal transition
(EMT) and the phenotype of breast cancer stem cells (CSCs). Vasculogenic mimicry (VM) is …

Vasculogenic mimicry (VM) is the formation of vascular channels lacking endothelial cells.
These channels are lined by tumor cells with cancer stem cell features, positive for periodic …

Vasculogenic mimicry (VM) refers to the unique capability of aggressive tumor cells to mimic
the pattern of embryonic vasculogenic networks. In the study we demonstrated that CD133 …

Vasculogenic mimicry (VM), a newly defined pattern of tumor microvascularization differs
from angiogenesis and vasculogenesis in its noninvolvement of endothelial cells, by which …