Background Childhood exposure to polycyclic aromatic hydrocarbons (PAHs) or lead (Pb) is
associated with epigenetic modifications. However, the effects of their co-exposures on IGF1 …

Polycyclic aromatic hydrocarbons (PAHs), as a kind of endocrine disruptors, can enter the
fetus body cross the placental barrier from prenatal PAHs exposure to cause adverse birth …

Informal e-waste recycling activities results in serious environmental pollution of PAHs. We
evaluated the body burden of 16 PAH congeners and potential health risks for children. A …

Background Associations between prenatal exposure to polychlorinated biphenyls (PCBs)
and reduced birth-size, and between DNA methylation of insulin-like growth factor-2 (IGF-2) …

Background Polycyclic aromatic hydrocarbons (PAH) are carcinogenic, neurotoxic
environmental pollutants generated during incomplete combustion of fossil fuel and other …

Background Exposure to atmospheric fine particle matter (PM 2.5) pollution and the
absorbed pollutants is known to contribute to numerous adverse health effects in children …

Epigenetic perturbations induced by environmental exposures at susceptible lifestages
contribute to disease development. Even so, the influence of early life and ongoing …

Background: Lead exposure during early development causes neurodevelopmental
disorders by unknown mechanisms. Epidemiologic studies have focused recently on …

Prenatal exposure to phthalates negatively affects the offspring's health. In particular,
epigenetic alterations, such as DNA methylation, may connect phthalate exposure with …

Background: Maternal factors are implicated in the onset of childhood asthma. Differentiation
of naïve CD4+ T lymphocytes into pro-allergic T-helper 2 cells induces interleukin (IL) 4 …