The c-Jun N-terminal kinases (JNKs) are members of a larger group of serine/threonine
(Ser/Thr) protein kinases from the mitogen-activated protein kinase family. JNKs were …

The c-Jun N-terminal kinases (JNKs), as members of the mitogen-activated protein kinase
(MAPK) family, mediate eukaryotic cell responses to a wide range of abiotic and biotic stress …

c-Jun N-terminal kinases (JNKs), first characterized as stress-activated members of the
mitogen-activated protein kinase (MAPK) family, have become a focus of inhibitor screening …

The literature provides strong precedent for both protumorigenic and tumor suppressor roles
for the c-Jun N-terminal kinases (JNKs) in the setting of oncogenesis. Clearly, JNKs are …

The c-Jun-NH 2-terminal kinase (JNK) signaling pathway plays a critical role in regulating
cell fate, being implicated in a multitude of diseases ranging from cancer to neurological and …

The c‐Jun N‐terminal kinases (JNKs) are members of the mitogen‐activated protein kinase
(MAPK) family. In mammalian genomes, three genes encode the JNK family. To evaluate …

The c-Jun NH2-terminal kinases (JNKs) are an evolutionarily conserved sub-group of
mitogen-activated protein (MAP) kinases. Recent studies have improved our understanding …

Different c-Jun N-terminal kinases (JNKs) are activated by a plethora of signals and
phosphorylate substrates such as c-Jun, which is required for efficient cell cycle progression …

The c-Jun N-terminal kinases (JNKs) are a subfamily of the mitogen-activated protein
kinases (MAPKs). Although progress in evaluating the functions of other MAPKs has been …

The regulation of c-Jun transcriptional activity by Jun N-terminal kinase (JNK) has become a
paradigm for understanding how mitogen-activated protein (MAP) kinase signalling …