Prenatal exposure to endocrine disruptors such as bisphenol A (BPA) plays a critical role in
the developmental programming of liver dysfunction that is characteristic of nonalcoholic …

Background: Maternal obesity increases offspring propensity to metabolic dysfunctions and
to non-alcoholic fatty liver disease (NAFLD), which may lead to cirrhosis or liver cancer. The …

Bisphenol A (BPA) is one of the most prevalent endocrine disrupting chemicals in the
environment. Developmental exposure to BPA is known to be associated with liver …

Developmental exposure to bisphenol A (BPA) is associated with liver dysfunction and
diseases in adulthood. The aims of this study were to assess the effects of prenatal BPA …

Bisphenol A (BPA) is a major environmental pollutant and food contaminant with endocrine-
disrupting effects on human and animal health. Perinatal and developmental exposure to …

Background: Exposure to chemicals during critical windows of development may re-program
liver for increased risk of nonalcoholic fatty liver disease (NAFLD). Bisphenol A (BPA), a …

Intrauterine growth restriction leads to the development of adult onset obesity/metabolic
syndrome, diabetes mellitus, cardiovascular disease, hypertension, stroke, dyslipidemia …

Our previous studies demonstrated that prenatal bisphenol A (BPA) exposure affected the
hepatic transcriptome and lipidome in rat offspring in a sex-and age-dependent manner. In …

The risk of obesity in adulthood is subject to programming beginning at conception. In
animal models, exposure to maternal obesity and high fat diets influences the risk of obesity …

Increasing evidence indicates that disturbance of the clock genes, which leads to systemic
endocrine perturbation, plays a crucial role in the pathogenesis of metabolic and liver …