In spinal cord injury (SCI), the initial damage leads to a rapidly escalating cascade of
degenerative events, known as secondary injury. Loss of mitochondrial homeostasis after …

Traumatic spinal cord injury exacerbates disability with time due to secondary injury
cascade triggered largely by overproduction of reactive oxygen species (ROS) at the lesion …

Spinal cord injury (SCI) produces excess reactive oxygen species (ROS) that can
exacerbate secondary injury and lead to permanent functional impairment. Hypothesizing …

Spinal cord injury (SCI) is damage to the spinal cord that leads to sudden loss of motor and
autonomic function and sensory under the level of the injury. The pathophysiological …

Mitochondrial dysfunction following spinal cord injury (SCI) may be critical for the
development of secondary pathophysiology and neuronal cell death. Previous studies have …

Mitochondrial dysfunction is a hallmark of secondary neuroinflammatory responses and
neuronal death in spinal cord injury (SCI). Even though mitochondria-based therapy is an …

Introduction Recently, the focus of oncological research has been on the optimization of
therapeutic strategies targeted at malignant diseases. Nanomedicine utilizing silicon dioxide …

Traumatic injury to the mammalian spinal cord is a highly dynamic process characterized by
a complex pattern of pervasive and destructive biochemical and pathophysiological events …

Spinal cord injuries (SCIs) are devastating. In SCIs, a powerful traumatic force impacting the
spinal cord results in the permanent loss of nerve function below the injury level, leaving the …

Background Iron oxide nanoparticles (IONPs) can attenuate oxidative stress in a neutral pH
environment in vitro. In combination with an external electromagnetic field, they can also …