Innate immune activation is essential to mount an effective antiviral response and to prime
adaptive immunity. Although a crucial role of CD169+ cells during vesicular stomatitis virus …

We have previously shown that mutations of CD14 or TLR4 impair type I interferon (IFN)
production and macrophage survival during infection with vesicular stomatitis virus (VSV) …

Long-term antigen expression is believed to play an important role in modulation of T-cell
responses to chronic virus infections. However, recent studies suggest that immune …

The effects of formalin on the infectivity and immunogenicity of vesicular stomatitis virus
(VSV) serotype Indiana were investigated. We found that formalin inactivation of VSV …

Vesicular stomatitis virus (VSV) is a prototypic nonsegmented negative-strand RNA virus.
VSV's broad cell tropism makes it a popular model virus for many basic research …

We have investigated virus-lymphocyte interactions by using cloned subpopulations of
interleukin-2-dependent effector lymphocytes maintained in vitro. Cloned lines of H-2 …

The soluble glycoprotein Gs of vesicular stomatitis virus (VSV), at approximately 10 (4)
molecules per cell, sensitized target cells for lysis by clones of CD4+ cytolytic T lymphocytes …

Vesicular stomatitis virus (VSV) is an insect-transmitted rhabdovirus that is neurovirulent in
mice. Upon peripheral VSV infection, CD169+ subcapsular sinus (SCS) macrophages …

Several studies indicated that TLR as well as retinoic acid–inducible gene I–like helicase
(RLH) signaling contribute to vesicular stomatitis virus (VSV)–mediated triggering of type I …

Background: Oncolytic vesicular stomatitis virus (VSV) can be delivered intravenously to
target primary and metastatic lesions, but the interaction between human peripheral blood …