Calcium overload is one of the early determinants of the core cellular events that contribute
to the pathogenesis of acute kidney injury (AKI), which include oxidative stress, ATP …

Acute kidney injury (AKI) is a common pathological process that is globally associated with a
high morbidity and mortality rate. The underlying AKI mechanisms include over-produced …

Calcium overload and ROS overproduction, two major triggers of acute kidney injury (AKI),
are self‐amplifying and mutually reinforcing, forming a complicated cascading feedback …

Early pathogenesis of ischemia-reperfusion (I/R)-induced acute kidney injury (AKI) is
dominated by intracellular calcium overload, which induces oxidative stress, intracellular …

Acute kidney injury (AKI) is a life‐threatening complication with a considerable occurrence
among patients. AKI is typically accompanied by an elevation in reactive oxidative species …

Acute kidney injury (AKI) is a common clinical disease with high morbidity and mortality, and
with a lack of effective drugs for treatment. Oxidative stress is very important in the …

Acute kidney injury (AKI) has been a global public health concern leading to high patient
morbidity and mortality in the world. Nanotechnology-mediated antioxidative therapy has …

Acute kidney injury (AKI) is frequently associated with reactive oxygen species (ROS) and
causes high mortality in clinics annually, and nanotechnology-mediated antioxidative …

Acute kidney injury (AKI) is a clinical syndrome that results from a rapid decline in renal
structure or renal functional impairment with the main pathological feature of sublethal and …

Acute kidney injury (AKI) is frequently triggered by abundant reactive oxygen/nitrogen
species (RONS) and leads to high morbidity and mortality in clinic. Unfortunately, the current …