Because neuronal nitric oxide synthase (nNOS) has a well-known impact on arteriolar blood
flow in skeletal muscle, we compared the ultrastructure and the hemodynamics of/in the …

We studied the relationship between neuronal NO synthase (nNOS) expression and
capillarity in the tibialis anterior (TA) muscle of mice subjected to treadmill training. The …

Vascular endothelial growth factor (VEGF) is considered to be important in promotion of
capillary growth in skeletal muscles exposed to increased activity. We studied its interactions …

During skeletal muscle contraction, NO derived from neuronal nitric oxide synthase (nNOS)
in skeletal muscle fibers or from endothelial cells (eNOS) may relax vascular smooth muscle …

Nitric oxide (NO) derived from endothelial NO synthase (eNOS) is an integral mediator of
vascular control during muscle contractions. However, it is not known whether neuronal …

Lau, Kim S., Robert W. Grange, Eiji Isotani, Ingrid H. Sarelius, Kristine E. Kamm, Paul L.
Huang, and James T. Stull. nNOS and eNOS modulate cGMP formation and vascular …

Key points• Neuronal nitric oxide (NO) synthase (nNOS) inhibition does not impact skeletal
muscle blood flow or vascular conductance (VC) during low‐speed (20 m min− 1) treadmill …

Vascular endothelial growth factor (VEGF) causes endothelial cell proliferation in vitro and
angiogenesis in vivo. Glycolytic skeletal muscles have a lower capillary density than …

Background Vascular endothelial growth factor (VEGF) mRNA levels increase in rat skeletal
muscle after a single bout of acute exercise. We assessed regional differences in VEGF 165 …

Nitric oxide (NO) has been shown to be a potent vasodilator released from endothelial cells
(EC) in large blood vessels, but NO release has not been examined in the capillary bed …