G protein-dependent signaling pathways control the activity of excitable cells of the nervous
system and heart, and are the targets of neurotransmitters, clinically relevant drugs, and …

G-protein-gated inwardly rectifying K+(GIRK) channels are essential effectors of inhibitory
neurotransmission in the brain. GIRK channels have been implicated in diseases with …

The G-protein activated, inward-rectifying potassium (K+) channels,“GIRKs”, are a family of
ion channels (Kir3. 1-Kir3. 4) that has been the focus of intense research interest for nearly …

G protein-coupled inwardly rectifying potassium (GIRK) channels are widely expressed
throughout the brain and mediate the inhibitory effects of many neurotransmitters. As a …

G protein-gated inwardly rectifying potassium (GIRK) channels are essential regulators of
cell excitability in the brain. While they are implicated in a variety of neurological diseases in …

G protein-gated inwardly rectifying K+ (GIRK/Kir3) channels exert a critical inhibitory
influence on neurons. Neuronal GIRK channels mediate the G protein-dependent …

G protein-activated inwardly rectifying K+(GIRK; Kir3) channels regulate the neuronal activity
and heart rate. Molecular cloning of the GIRK channel genes has led to remarkable progress …

G-protein–gated inwardly-rectifying K+(GIRK) channels are targets of G i/o-protein–signaling
systems that inhibit cell excitability. GIRK channels exist as homotetramers (GIRK2 and …

G protein-gated inwardly rectifying potassium channels (Kir3/GirK) are important for
maintaining resting membrane potential, cell excitability and inhibitory neurotransmission …

G protein-coupled inward rectifier K+ (GIRK) channels represent novel targets for the
development of new therapeutic agents. GIRK channels are activated by a large number of …