SNAIL is a key transcriptional regulator in embryonic development and cancer. Its effects in
physiology and disease are believed to be linked to its role as a master regulator of …

By fostering cell commitment to the epithelial-to-mesenchymal transition (EMT), SNAIL
proteins endow cells with motility, thereby favoring the metastatic spread of tumor cells …

The Snail zinc-finger transcription factors trigger epithelial-mesenchymal transitions (EMTs),
endowing epithelial cells with migratory and invasive properties during both embryonic …

Snail1 is a zinc finger transcriptional repressor whose pathological expression has been
linked to cancer cell epithelial–mesenchymal transition (EMT) programs and the induction of …

The transcription factor Snail1 induces epithelial-to-mesenchymal transition (EMT) in tumor
epithelial cells, a process associated with the emergence of stemness, invasion and cancer …

Snail is primarily known as a transcriptional repressor that induces epithelial-mesenchymal
transition by suppressing adherent proteins. Emerging evidence suggests that Snail can act …

Snail has moved into the fast lane of development and cancer biology with the epithelial-
mesenchymal transition (EMT) emerging as one of the hottest topics in medical science …

The zinc finger transcription factor Snail is a known effector of epithelial-to-mesenchymal
transition (EMT), a process that underlies the enhanced invasiveness and chemoresistance …

The zinc-finger transcription factor Snail is believed to trigger epithelial–mesenchymal
transitions (EMTs) during cancer progression. This idea is supported by analysis of Snail …

Expression of the EMT-inducing transcription factor Snail is enhanced in different human
cancers. To investigate the in vivo role of Snail during progression of epithelial cancer, we …