After spinal cord injury, transected axons fail to regenerate, yet significant, spontaneous
functional improvement can be observed over time. Distinct central nervous system regions …

Traumatic spinal cord injury (SCI) is a detrimental condition that causes loss of sensory and
motor function in an individual. Many complex secondary injury cascades occur after SCI …

Young rats display an accelerated rate of locomotor recovery after contusive spinal cord
injury (SCI) compared to adults subjected to a similar standardized injury. We examined …

After spinal cord injury (SCI), about 50% of the oligodendrocytes and astrocytes in the
residual white matter at the injury site are lost by 24 h. However, chronically after SCI, the …

Studies in the rat have shown that contusive spinal cord injury (SCI) results in devastating
pathology, including significant loss of mature oligodendrocytes and astrocytes even in …

The adult mammalian spinal cord contains neural stem and/or progenitor cells that slowly
multiply throughout life and differentiate exclusively into glia. The contribution of adult …

Harnessing the regenerative capabilities of endogenous precursor cells in the spinal cord
may be a useful tool for clinical treatments aimed at replacing cells lost as a consequence of …

Abstract Background Context The most common causes of spinal cord injury (SCI) are
traumatic traffic accidents, falls, and violence. Spinal cord injury greatly affects a patient's …

Spinal cord injury (SCI) results in motor and sensory deficits, the severity of which depends
on the level and extent of the injury. Animal models for SCI research include transection …

The secondary loss of neurons and glia over the first 24 h after spinal cord injury (SCI)
contributes to the permanent functional deficits that are the unfortunate consequence of SCI …