Erythrocyte invasion by Plasmodium falciparum is central to the pathogenesis of malaria.
Invasion requires a series of extracellular recognition events between erythrocyte receptors …

Invasion of host erythrocytes is essential to the life cycle of Plasmodium parasites and
development of the pathology of malaria. The stages of erythrocyte invasion, including initial …

Plasmodium falciparum is the parasite responsible for the most lethal form of malaria, an
infectious disease that causes a large proportion of childhood deaths and poses a …

Plasmodium falciparum causes the severe form of malaria that has high levels of mortality in
humans. Blood-stage merozoites of P. falciparum invade erythrocytes, and this requires …

The members of the phylum Apicomplexa parasitize a wide range of eukaryotic host cells.
Plasmodium falciparum, responsible for the most virulent form of malaria, invades human …

Invasion of erythrocytes by Plasmodium falciparum merozoites is a complex multi-step
process mediated by specific interactions between host receptors and parasite ligands …

The malaria parasite, Plasmodium falciparum, exploits multiple ligand-receptor interactions,
called invasion pathways, to invade the host erythrocyte. Strains of P. falciparum vary in their …

Multiple interactions between parasite ligands and their receptors on the human erythrocyte
are a condition of successful Plasmodium falciparum invasion. The identification and …

Plasmodium falciparum, the cause of almost all human malaria mortality, is a member of the
Laverania subgenus which infects African great apes. Interestingly, Laverania parasites …

Plasmodium falciparum causes the most severe form of malaria in humans and invades
erythrocytes using multiple ligand-receptor interactions. Two important protein families …