Ischemic damage to the brain and loss of neurons contribute to functional disabilities in
many stroke survivors. Recovery of neuroplasticity is critical to restoration of function and …

Cerebral ischemia may cause irreversible neural network damage and result in functional
deficits. Targeting neuronal repair after stroke potentiates the formation of new connections …

The nervous and immune systems control whole-body homeostasis and respond to various
types of tissue injury, including stroke, in a coordinated manner. Cerebral ischaemia and …

Traumatic brain injury (TBI) is one of the leading causes of both acute and long-term
morbidity in the pediatric population, leading to a substantial, long-term socioeconomic …

Stroke is the world's leading cause of disability with limited brain repair treatments which
effectively improve long‐term neurological deficits. The neuroinflammatory responses …

Recent work has demonstrated that self-repair in the adult brain can be augmented by the
infusion of growth factors to activate endogenous neural precursor cells that contribute to …

Non-invasive criteria determining the progress of brain healing are especially important in
aging, providing a case-specific therapeutic strategy in populations with dysregulated …

The injured brain can be stimulated to amplify its intrinsic restorative processes to improve
neurological function. Thus, after stroke, both cell and pharmacological neurorestorative …

Replacement of dead neurons following ischemia, either via enhanced endogenous
neurogenesis or stem cell therapy, has long been sought. Unfortunately, while various …

Following a cerebral ischemic event, substantial alterations in both cellular and molecular
activities occur due to ischemia-induced cerebral pathology. Mounting evidence indicates …