Herpesviral infection reflects thousands of years of coevolution and the constant struggle
between virus and host for control of cellular gene expression. During Kaposi's sarcoma …

One striking characteristic of certain herpesviruses is their ability to induce rapid and
widespread RNA decay in order to gain access to host resources. This phenotype is induced …

During Kaposi's sarcoma (KS)–associated herpesvirus (KSHV) lytic infection, many virus-
encoded signaling molecules (eg, viral G protein–coupled receptor [vGPCR]) are produced …

Kaposi's sarcoma-associated herpesvirus (KSHV) induces life-long infections and has
evolved many ways to exert extensive control over its host's transcriptional and post …

The Kaposi's sarcoma-associated herpesvirus (KSHV) alkaline exonuclease SOX, encoded
by open reading frame 37 (ORF37), is a bifunctional early-lytic-phase protein that possesses …

Kaposi's sarcoma-associated herpesvirus (KSHV) is an oncogenic γ-herpesvirus that infects
humans and exhibits a biphasic life cycle consisting of latent and lytic phases. Following …

Kaposi's sarcoma-associated herpesvirus (KSHV), like other herpesviruses, replicates within
the nuclei of its human cell host and hijacks host machinery for expression of its genes. The …

The Kaposi's sarcoma-associated herpesvirus (KSHV) SOX protein, encoded by ORF37,
promotes shutoff of host cell gene expression during lytic viral replication by dramatically …

The life cycle of Kaposi's sarcoma-associated herpesvirus (KSHV) consists of two phases,
latent and lytic. The virus establishes latency as a strategy for avoiding host immune …

Nuclear RNAs are subject to a number of RNA decay pathways that serve quality control
and regulatory functions. As a result, any virus that expresses its genes in the nucleus must …