There are different BCR-ABL1 fusion genes that are translated into proteins that are different
from each other, yet all leukemogenic, causing chronic myeloid leukemia (CML) or acute …

Background and objective: The characteristic chromosomal translocation t (9; 22)(q34; q11)
in chronic myeloid leukaemia (CML) mainly results in the two different BCR/ABL fusion …

To explore the type, prevalence and outcomes in chronic myeloid leukaemia (CML) patients
with uncommon BCR‐ABL 1 transcripts in the era of tyrosine kinase inhibitors (TKI s) …

The incidence of one or other rearrangement in chronic myeloid leukemia (CML) patients
varies in different reported series. In this study we report the frequencies of BCR-ABL1 …

Background: A specific chromosomal abnormality, the Philadelphia chromosome, is present
in 90–95% of patients with CHRONIC MYELOID LEUKEMIA. The aberration results from a …

BACKGROUND AND OBJECTIVES: The most common translocation in chronic myeloid
leukemia (CML) t (9; 22)(q34; q22) produces the BCR/ABL fusion gene. We set up and …

Chronic myeloid leukemia (CML) is characterized by the presence of BCR-ABL1 fusion
gene. In over 95% of CML patients, the typical BCR-ABL1 transcript subtypes are e13a2 …

A novel variant of the chimeric BCR-ABL mRNA transcript was detected in a patient with
Philadelphia chromosome-negative (Ph-) chronic myelogenous leukemia (CML) by …

Background Kinase domain mutations in BCR-ABL1 are associated with resistance to
tyrosine kinase inhibitors in patients with chronic myeloid leukaemia. Next-generation …

The most common breakpoint cluster region gene-Abelson murine leukemia viral oncogene
homolog 1 (BCR-ABL) transcripts in chronic myeloid leukemia (CML) are e13a2 (b2a2) and …