Helicobacter pylori colonization induces inflammation in essentially all hosts, a persistent
process that increases the risk of developing distal gastric adenocarcinoma. However, only …

Helicobacter pylori cag+ strains enhance gastric epithelial cell proliferation and attenuate
apoptosis in vivo, which may partially explain the increased risk of gastric cancer associated …

Helicobacter pylori (H. pylori) infection is the major risk factor associated with the
development of gastric cancer. The transition from normal mucosa to non-atrophic gastritis …

The role of Helicobacter pylori in gastric carcinogenesis is supported almost exclusively by
epidemiological data and prospective histopathological studies. From biological and …

Helicobacter pylori was definitively identified by culture in 1984 by Robin Warren and Barry
Marshall, 1 and 10 years later this organism was recognized by the International Agency for …

Gastric adenocarcinoma is the second most common cause of cancer-related mortality
worldwide. Infection with Helicobacter pylori is the single most common cause of …

Background & Aims: Human colonization with Helicobacter pylori increases the risk for distal
gastric adenocarcinoma, possibly by altering gastric epithelial cell cycle events and/or …

Molecular mechanisms of gastric cancer initiation and progre... : Current Opinion in Infectious
Diseases Molecular mechanisms of gastric cancer initiation and progression by Helicobacter …

Although gastric adenocarcinoma is associated with the presence of Helicobacter pylori in
the stomach, only a small fraction of colonized individuals develop this common malignancy …

Colonization of the human stomach by Helicobacter pylori and its role in causing gastric
cancer is one of the richest examples of a complex relationship among human cells …