Myocardial infarction triggers an inflammatory cascade that results in healing and
replacement of the damaged tissue with scar. Cardiomyocyte necrosis triggers innate …

Healing of myocardial infarcts depends on an inflammatory cascade that ultimately results in
clearance of dead cells and matrix debris and formation of a scar. Myocardial necrosis …

Myocardial necrosis triggers an inflammatory reaction that clears the wound from dead cells
and matrix debris, while activating reparative pathways necessary for scar formation. A …

Acute myocardial infarction (MI) occurs when blood flow to the myocardium is restricted,
leading to cardiac damage and massive loss of viable cardiomyocytes. Timely restoration of …

In adult mammals, massive sudden loss of cardiomyocytes after infarction overwhelms the
limited regenerative capacity of the myocardium, resulting in the formation of a collagen …

The wound healing response after myocardial infarction (MI) involves a cascade of
molecular and cellular events that lead to a replacement of the necrotic area with a collagen …

The dynamic alterations in the cardiac extracellular matrix following myocardial infarction not
only determine the mechanical properties of the infarcted heart, but also directly modulate …

Myocardial infarction is defined as sudden ischemic death of myocardial tissue. In the
clinical context, myocardial infarction is usually due to thrombotic occlusion of a coronary …

Myocardial infarction triggers an intense inflammatory response that is essential for cardiac
repair, but which is also implicated in the pathogenesis of postinfarction remodelling and …

Myocardial infarction is associated with an inflammatory response, ultimately leading to
healing and scar formation. Reperfused myocardial infarcts exhibit an enhanced …